VEGF-D promotes tumor metastasis by regulating prostaglandins produced by the collecting lymphatic endothelium.

نویسندگان

  • Tara Karnezis
  • Ramin Shayan
  • Carol Caesar
  • Sally Roufail
  • Nicole C Harris
  • Kathryn Ardipradja
  • You Fang Zhang
  • Steven P Williams
  • Rae H Farnsworth
  • Ming G Chai
  • Thusitha W T Rupasinghe
  • Dedreia L Tull
  • Megan E Baldwin
  • Erica K Sloan
  • Stephen B Fox
  • Marc G Achen
  • Steven A Stacker
چکیده

Lymphatic metastasis is facilitated by lymphangiogenic growth factors VEGF-C and VEGF-D that are secreted by some primary tumors. We identified regulation of PGDH, the key enzyme in prostaglandin catabolism, in endothelial cells of collecting lymphatics, as a key molecular change during VEGF-D-driven tumor spread. The VEGF-D-dependent regulation of the prostaglandin pathway was supported by the finding that collecting lymphatic vessel dilation and subsequent metastasis were affected by nonsteroidal anti-inflammatory drugs (NSAIDs), known inhibitors of prostaglandin synthesis. Our data suggest a control point for cancer metastasis within the collecting lymphatic endothelium, which links VEGF-D/VEGFR-2/VEGFR-3 and the prostaglandin pathways. Collecting lymphatics therefore play an active and important role in metastasis and may provide a therapeutic target to restrict tumor spread.

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عنوان ژورنال:
  • Cancer cell

دوره 21 2  شماره 

صفحات  -

تاریخ انتشار 2012